AB081. Research on epididymis inflammatory mass in 1,021 patients with varicocele

نویسندگان

  • Rui Wang
  • Tian-Biao Zhang
  • Tao Zheng
  • Rui Li
  • Chao-Liang Wang
  • Fan Yang
  • Donghui Jia
  • Jing Lian
  • Wei-Xing Zhang
چکیده

© Translational Andrology and Urology. All rights reserved. Transl Androl Urol, 2017;6(S3) tau.amegroups.com 17α-hydroxylase were examined by western blotting and RT-PCR, respectively. Annexin A5 significantly stimulated testosterone secretion from rat Leydig cells in doseand time-dependent manners and increased mRNA and protein expression of StAR, P450scc, 3β-HSD and 17β-HSD but not 17α-hydroxylase. Annexin A5 knockdown by siRNA significantly decreased the level of testosterone and protein expression of P450scc, 3β-HSD and 17β-HSD. The significant activation of ERK1/2 signaling was observed at 5, 10, and 30 min after annexin A5 treatment. After the pretreatment of Leydig cells with ERK inhibitor PD98059 (50 μmol/L) for 20 min, the effects of annexin A5 on promoting testosterone secretion and increasing the expression of P450scc, 3β-HSD and 17βHSD were completely abrogated (P<0.05). Thus, ERK1/2 signaling is involved in the roles of annexin A5 in mediating testosterone production and the expression of P450scc, 3β-HSD and 17β-HSD in Leydig cells.

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عنوان ژورنال:

دوره 6  شماره 

صفحات  -

تاریخ انتشار 2017